简介：Thispaperanalysestheanteriordescendingbranchofthearteriacoronariasinistrafromtheviewofhemodynamics.Theresultsshowthatthebranchingoftheanteriordescendingbranchwithanangleof90°attheopeningandtheformationofthemuralarteriacoronariaarethehemodynamiccharacteristics.Theshearingstressoftheturbulentflow,theadditionalpressure,theformationofthebloodvertex,andtheimpulsivepressure,causedbythesecharacteristics,arehemodynamicreasonsthatleadstotheoccurenceofatherosclerosisinanteriordescendingbranch.

简介：TheundecapeptidesubstanceP(SP)wasshowntobeintimatelyinvolvedinboththestructuralandfunctionalaspectsoftheanteriorpituitary.Yet,inadditiontoitsinfluencesonhormonalsecretion,SPmaywellpossessmoreactionsinthismastergland.ThepresentstudywasfthereforeaimedtoinvestigatetheeffectofSPontheproliferationofratanteriorpituitarycellsinprimaryculture,ItwasfoundthatSPcoulddose-dependentlyincreasetheincorporationoftritiatedthymidine(3H-TdR)intoculturedanteriorpituitarycells.OthermammaliantachykininssuchasneurokininAandneurokininBhadsimilareffectbuttovaryingdegrees.TheequipotentanalogueofSP,Norleucine^11-SP(Nle^11-SP),alsoactedlikewise.withitsactionantagonizablebyspantide,aSPreceptorblocker.TofurthercharacterizethenatureofcellsresponsivetothechallengeofSP,immunocytochemicalstainingagainstS-100proteinandsomeadenohypophysealhormoneswasperformedaloneorplusautoradiography.TheresultsshowedthatthepercentageofS-100proteinimmunorectivecellswasapparentlyelevatedbytheaddtionofNle^11-Spfor48h,whichindicatesapreferentialproliferationoffolliculo-stellatecellsundertheregime.ThiswasconfirmedbyincreasesinimmunocytochemicalorautoradiographicallabellingindicesofanteriorpituitarySubstancePandanteriorpituitarycellproliferation.Cellstreatedsimilarly.Takentogether,TheseresultsrevealthatthetrophicactionofSPobservedpreviouslyinothertissuesisalsopresentatleastinculturedratanteriorpituitarycells.withrespondingcellsbeingpredominantlyfolliculo-stellatecellsastypifiedbyS-100proteinimmunoreactivity.Therefore,anintra-pituitarytrophicactionofSPinvivocouldbeanticipated.

简介：Objective:Toanalyzethehistologicalresultsandthebiologicalremodelingofligamentousinsertionafterthereconstructionofanteriorcruciateligament(ACL)withautograftorallografttendon.Methods:ExtensordigitorumtendonwasharvestedfromhindlimbasgraftmaterialandtransplantedtoreconstructtheresectedACLin12mongreldogs.Eachfreetendonendwassecuredbyholdingsuturesandthenthesuturesweretiedtothepostscrewatthefemoralandtibialbonytunneloutletaftertransplantationrespectively.Autograftwasrandomlyperformedononesideofkneewhileallograftontheothersideofknee.Aftertransplantation,thehistologicalanalysiswasundertakenatthe6th,12thweeksandthe6thmonthusinghematoxylin-eosin(HE)stainunderlightmicroscope.Results:TheinsertionstructureofnormalACLtypicallyconsistedoffourlayers,i.e.,denseconnectivetissue,fibrocartilage,mineralizedfibrocartilageandbone.Therewasadistinctregulartidemarklinebetweenfibrocartilageandmineralizedfibrocartilage.Atthe6thweekpostoperativcly,looseconnectivetissuepresentedintheinterspacebetweengraftandbonytunnelwallinbothautograftandallograftgroups.Atthe12thweekpostoperatively,thecollagenousfibersbetweenautograftandtunnelwallbecamewellorganizedandthefourlayersofinsertionwithdiscontinuoustidemarklineweredemonstratedindistinctlyinautograftgroupbutnotinallograftgroup.Atthe6thmonthpostoperatively,bothofaclearandcontinuoustidemarklineanddistinctfourlayerscouldbeseeninautograftgroup.Inallograftgroup,onlyawaveddiscontinuoustidemarklinewasshownandeithertheanatomicmorphologyorthematurityofinsertionwasinferiortothatofautograftgroup.Conclusions:Atthe6thmonthpostoperatively,althoughtheligament-cartilageinsertionisprimarilyformedafterACLreconstructionwithautograftorallografttendon,thehistologicalmorphologyandthematurationofinsertionofautografttendonar

简介：Injuryofankleisthemostcommoninathleticinjury.Becauseofanatomicparticu-larityofanklejoint,i.e.lateralmalleolusislongandthinandislocatedattheposteriorandlowerthanmedialmalleolus.Themedialmalleolusisbroadandflatandislocatedattheanterior.Lateralligamentisthinnerandweakerthanmedialligamentandthemusclegroupssupportingstrephexopodiaisfarinferi-ortothemusclegroupssupportingstrephenopodiainstrength,injuryofanklejointhappensmoreatinversionsprainandin-juryofanteriortalofibularligamentisinducedoften.Thus,rapidandeffectivetreatmentofanteriortalofibularligamentinjurycanelevatebetterathleticsofsportsman.Wheneverthediseasehappenedinstudentstoexercise,theauthorsoftenusedacupunctureofQiuxu（GB40）-through-Zhaohai（KI6）pointwithstrongstimulativemanipulation,whichcouldrelievemoreremakedlypainoftheanklejointcausedbyanteriortalofibularligamentinjuryasre-portedinthe