摘要
BackgroundApulmonary-infectedpatienthadseverehyponatremiathatlastedtwodaysduringhospitalization.Wecouldnotfindanyhistorysuchasgluttony,liverandgalldisease.Theexaminationofliverfunctionreportedthatthehepaticenzymologyisnormal.InICU,wequicklyfoundhemodiastasereachedatextremelyhighlevel.TheberryCTindicatedthattheheadofpancreasswellingandtheboundarywasobscureandeffusive.Thepatientwasdiagnosedasacutepancreatitis.Thiscaseraisesaquestiontous:Doesseverehyponatremiacauseacutepancreatitis?Ithasnotbeenreported.MethodsWeestablishedacuteanimalmodelofhyponatremiathatdidnotincludeanyotherelectrolytechaos.Aftertheanimalmodelofhyponatraemiacompleted,serumamylaselevelsandpathologicalexaminationwereanalyzed.ResultsThehyponatremiamodels,outof16rats,onlyfourrats'serumsodiumwerelessthan120mmol/L.Inthesefourrats,besidesserumsodium,theremainingelectrolyteswerenormalandserumlipidswereinnormalrange,onlyonerat'sserumamylaseincreasedsignificantlymorethanfourtimesofthenormalcontrolgroupanditspathologicalfindingshintedpancreaticedema.ConclusionsTheexperimentalmodelsuccessfullyduplicatedtheclinicalandpathologicfeaturesofpancreatitisinducedbyhyponatraemia.Throughthiscasereport,wehopethatattentioncanbepaidtochangesinserumamylasewhenhyponatremiaappeared.
出版日期
2010年03月13日（中国期刊网平台首次上网日期，不代表论文的发表时间）
