Oligomerization of Vibrio cholerae Hemolysin Induces CXCR3 Upregulation and Activation of B-1a Cell-中国期刊网

摘要 hemolysinoligomer支持B-1a房间和CD25的表示的增长，它房间激活是指示的，在B-1a房间上。当单体没能导致这些效果时，oligomer导致的CD86的upregulation为B7家庭的B7-2成员显示出它的选择偏爱。oligomer导致了CXCR3的表示，当单体导致了CXCL4的表示时，与B房间激活联系了，强大的angiostaticchemokine。在结论，我们发现了房间由表示CXCL4回答了到apoptogenic单体的那B-1a，而immunogen的oligomerization导致了CXCR3向激活转移反应。

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Oligomerization of Vibrio cholerae Hemolysin Induces CXCR3 Upregulation and Activation of B-1a Cell

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Oligomerization of Vibrio cholerae Hemolysin Induces CXCR3 Upregulation and Activation of B-1a Cell

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