简介:TheNorthYellowSeaBasin(NYSB),whichwasdevelopedonthebasementofNorthChina(Huabei)continentalblock,isatypicalcontinentalMesozoic-Cenozoicsedimentarybasinintheseaarea.ItsMesozoicbasinisaresidualbasin,belowwhichthereisprobablyalargerPaleozoicsedimentarybasin.TheNorthYellowSeaBasincomprisesfoursagsandthreeuplifts.Ofthem,theeasternsagisaMesozoic-CenozoicsedimentarysaginNYSBandhasthebiggestsedimentthickness;thecurrentKoreandrillingwellsareconcentratedintheeasternsag.Thissagiscomparativelyrichinoilandgasresourcesandthushasarelativelygoodpetroleumprospectinthesea.ThecentralsaghasalsoaccommodatedthickMesozoic-Cenozoicsediments.ThelatestresearchresultsshowthattherearethreeseriesofhydrocarbonsourcerocksintheNorthYellowSeaBasin,namely,blackshalesofthePaleogene,JurassicandCretaceous.TheprincipalhydrocarbonsourcerocksinNYSBaretheMesozoicblackshale.AccordingtothedrillingdataofKorea,theblackshalesofthePaleogene,JurassicandCretaceoushaveallcomeuptothestandardsofgoodandmaturesourcerocks.TheNYSBownsanintactsystemofoilgeneration,reservoirandcappingrocksthatcanhelphydrocarbontoforminthebasinandthusithasthegreatpotentialofoilandgas.TheverticaldistributionofthehydrocarbonresourcesismainlyconsideredtobeintheCretaceousandthenintheJurassic.
简介:<正>InOctoberofthisyear,adesignevaluationmeetingonfeasibilitystudyofUHVDCtransmissionprojectwaspresidedoverbytheStateGridCorporation.Itindicatesthatthecorporationmadeagreatprogressinthepre-engineeringworkofUHVDCtransmission.TheUHVDCtransmissionprojects,whichsendelectricityfromJinshaRiverPhaseI(XiluoduandXiangjiabahydropowerstations)totheCentralandEasternChinaandfromJinping
简介:Aim:Tostudytheeffectofdiabetesmellitusandinsulintreatmentonratpenilenitricoxidesynthasecontent.Methods:MaleWistarratsweredividedatrandomintotwogroups:theControl(n=8)andtheDiabetic(n=17).Diabetesmellituswasinducedbyintraperitonealinjectionofstreptozotocin.Thediabeticanimalswerethenrandomlydividedintotwosubgroups:diabeticratswithoutinsulintreatment(n=7)anddiabeticratswithinsulintreatment(n=10).Theneuronalnitricoxidesynthase(nNOS)inthepenilecorpuscavemosumwereassayedbyimmumohistochemicalstainingwithspecificantibodytonNOSandthenNOS-positivenervefiberswerecountedsemiquantitativelyunderahighpowermicroscope.Results:ThenNOS-positivenervefibresindiabeticratswithtreatmentwashigherthanthatindiabeticratswithouttreatment(P<0.05)andlowerthanthatinthecontrols(P<0.01).ThenNOS-positivenervefibresindiabeticratwithouttreatmentwerealsolowerthanthatinthecontrols(P<0.01).Conclusion:Instreptozotocin-induceddiabeticrats,thenNOScontentinthepenilecorpuscavemosumwassignificantlydecreased.InsulintreatmentatthedoselevelemployedpartiallyrestoresthepenilenNOScontentintheserats.
简介:Insufficientgrowthandrarefactionofcapillaries,followedbyendothelialdysfunctionmayrepresentoneofthemostcriticalmechanismsinvolvedinheartdamage.Inthisstudyweexaminedhistochemicalandultrastructuralchangesinmyocardialcapillaryendotheliumintwomodelsofheartfailurestreptozotocin-induceddiabetesmellitus(STZ)andNOdeficienthypertensioninmaleWistarrats.Diabeteswasinducedbyasinglei.v.doseofSTZ(45mg/kg)andchronic9-weekstagewasanalysed.ToinduceNO-deficienthypertension,animalsweretreatedwithinhibitorofNOsynthaseLnitroargininemethylester(L-NAME)(40mg/kg)for4weeks.Leftventriculartissuewasprocessedforenzymecatalytichistochemistryofcapillaryalkalinephosphatase(AlPh),dipeptidylpeptidaseⅣ(DPPⅣ),andendothelialNOsynthase/NADPH-diaphorase(NOS)andforultrastructuralanalysis.Indiabeticandhypertensiverats,lower/absentAlPhandDPPⅣactivitieswerefoundinfocalmicro-areas.NOSactivitywassignificantlyreducedandpersistedonlylocally.QuantitativeevaluationdemonstratedreductionofreactionproductintensityofAlPh,DPPandNOSby49.50%,74.36%,20.05%indiabeticand62.93%,82.71%,37.65%inhypertensiverats.Subcellularalterationsofendothelialcellswerefoundinheartofbothgroupssuggestinginjuryofcapillaryfunctionaswellascompensatoryprocesses.Endothelialinjurywasmoresignificantindiabeticanimals,incontrasttheadaptationwasmoreevidentinhypertensiveones.Concluding:bothSTZ-induceddiabetes-andNO-deficienthypertension-relatedcardiomyopathywereaccompaniedbysimilarfeaturesofstructuralremodellingofcardiaccapillarynetworkmanifestedasangiogenesisandangiopathy.Thelatterwashowever,predominantandmayacceleratedisappearanceofcapillaryendotheliumcontributingtomyocardialdysfunction.
简介:Inthispaper,anewdevelopedmulti-channelpre-processingcircuitforsignalsfromtemperaturesensorwasintroducedinbrief.Thiscircuitwasdevelopedtocollectandamplifythesignalsfromtemperaturesensor.Thisisauniversalcircuit.Itcanbeusedtoprocessthesignalsfromthermocouplesandalsousedtoprocesssignalsfromthermistors.Thiscircuitwasmountedinastandardbox(440W×405D×125Hmm)asaninstrument.The
简介:在现在的学习,胰腺的小岛房间的ultramicrostructure的fasting血葡萄糖(FBG)集中和浆液胰岛素集中和chenges被用作这些索引在试验性的糖尿病mellitus(DM)的处理评估aurioular针灸的治疗学的效果并且在兔子.Thirty分析ist机制--二只兔子随机被划分成控制,模型,针灸和药有在每个group.DM模型的8个盒子的组被建立由静脉内两个组的处理每天被给一次,20个会议完全。在处理(20个会议)的2堂功课以后,与预告的处理和模型组比较,FBG在针灸和药groupsd满足的结果显示显著地减少了(P0.01),当胰岛素显著地针灸和药组increaesd满足时(P0.05)。在FBG和胰岛素内容的针灸和药组之间的没有重要差别。因此,显微镜的dbservation显示出的电子在Iysosomebeing的部分organells的一些改进少数,核仁的胀大的减小,在两个acupucture组的等等和药组。它显示出胰的小岛房间的结构的那恢复,合成的增加和胰岛素以及胰岛素mayy的活动的版本在DM兔子在lowring血葡萄糖水平贡献耳的针灸的效果。
简介:Albiziasaman(Jacq)F。Muell。通常作为雨知道树种子与五预先播种被对待学习预先播种的效果的处理在托儿所的萌芽和起始的幼苗开发上的处理。实验在林地和环境科学的研究所的托儿所被建立,Chittagong的大学,Chittagong,孟加拉国。结果揭示了在种子的一个方面抓紧的那根钉子(在theseed的远侧的结束)(T_4)提供(50%)最高播种萌芽。第二最高的萌芽(42%)为为24h(T_1)在冷水里与沉浸对待的种子被获得。当为30s在煮的水里沉浸的种子在为24h浸泡的冷水里列在后面时,萌芽完全被禁止。另外的萌芽参数和起始的词法生长和幼苗的生物资源生产与控制(T_0)相比为处理T_4和T_1也是更高的处理。例如钉子在一个抓紧的T_4的预先播种的处理种子站在一起(在种子的远侧的结束)并且T_1(为24h在冷水里沉浸的种子)可以在托儿所为最大的萌芽和Albiziasaman的起始的精力旺盛的幼苗生长被推荐。
简介:MidwivesintheWellingtonregionhavenotedthatwomenreceivingprebirthacupunctureconsistentlyexperiencemoreefficientlabours.Midwivesreportthatthelengthoflabourisreduced,particularlyinprimigravidwomen,withareductioninmedicalinterventionrequired.Thispaperwillevaluateanauditof15midwiveswhohavecompletedaspecializedtrainingprogrammeinacupunctureandcurrentlyofferprebirthacupunctureasanadjuncttotheirmidwiferypractice.
简介:ObjectivesTostudyclinicalandcoronaryangiographicfindingsinpatientswithbothcoronaryheartdiseases(CHD)andtype2diabetesmellitus(T2DM).Methods215patientswithCHDconfirmedbycoronaryangiographywereinvolvedinthisstudy.Thepatientsweredividedintotwogroups:74CHDpatientswithT2DM(meanage64.7±8.2years,male/female47/27),and141CHDpatientswithoutT2DM(meanage66.2±9.2years,male/female100/41).Theclinicalfeaturesandthedatafromselectivecoronaryangiographieswerecomparedbetweentype2diabeticandnon-diabeticCHDpatients.ResultsComparedtonon-diabeticCHDpatients,thepatientswithbothCHDandT2DMsufferedmorefromacutemyocardialinfarction,silentischemiaandseverearrhythmias(P<0.01,P<0.05),andhadhigherserumtriglyceridesandapo-lipoproteinB,alongwithincreasedserumuricacid(P<0.01,P<0.05),increasedleftventricularenddiastolicdiameter(P<0.01),anddecreasedleftventricularejectionfraction(P<0.001).Comparedtonon-diabeticCHDpatients,thepatientswithbothCHDandT2DMsufferedmorefromtriplevesseldisease(P<0.01),severecoronaryarterystenosis,completeocclusionsanddiffuselesions(P<0.001).ConclusionsSevereclinicalmanifestation,leftventriculardysfunction,diffuseorcomplicatedlesionsofcoronaryarteriesweremorecommoninpatientswithbothCHDandT2DM,itsuggeststhatthetype2diabeticCHDpatientshavepoorprognosis.
简介:TheeffectofPentoxifylline(PTX)ontype1diabeteswasinvestigatedbymeansofthestudiesontheexpressionsofcytokinemRNAinpancreasandtheFas-Fasl,onisletcellsofNODmice.NODmiceweretreatedwithFFXfrom4-6wk,andthenfrom8-12wk.Aftertreatment,itwasfoundthattheincidenceofdiabetesinNODmiceatagesof30wkwasreducedto25%ingroupofmicetreatedwithPTX,incomparisonto73.3%incaseofmiceinjectedwithPBS,andthedegreeofinsulitisinthePTXtreatedmicewaslowerthanthatofthePBSinjectedmice.RT-PCRanalysisrevealeddown-regulatoryeffectontheexpressionsofIFN-γandTNF-αmRNAinPTXtreatedmice,buttherewasnoanyeffectontheexpressionofIL-10.AstotheexpressionofFas,therewasmarkeddecreaseinthemeancytoplasmicintegralopticaldensity(IOD)inPTXtreatedmice,buttherewaslittledifferencebetweenFIXandPBSgroupsintheexpressionofFasL.TheseresultsindicatedthatFIXcouldpreventthedevelopmentofdiabetesinNODmice,whichmightberelatedtotheregulationofTh1/Th2imbalanceandthedecreasedexpressionofFasinisletcells.
简介:Twograftingmethods,i.e.solutiongraftingandpre-adsorbing,areintroducedtocovalentlyimmobilizehydrophilicpolymersonsegmentedpolyurethane(SPU)tomodifyitshydrophilicityandtoimproveitscellcompatibility.Solutiongraftingresultsinhigherdegreeofgraftingandroughersurfacemorphology.Cellcultureevaluationdemonstratesthatthemodifiedmembranesthusobtainedaredisadvantageoustotheendothelialcell(ECs)growthprobablybecausethehydrophilicgroupsonthesurfaceareover-crowded.However,pre-absorbinggraftinggenerateslowerdegreeofgrafting,whichisdetectedandconfirmedbyATR-FTIRspectraandwatercontactangle.Scanningelectronmicroscopic(SEM)measurementshowsthatthelattermethodproducesaplaneandsmoothmorphology,whichissimilartotheSPUcontrolsamplecontrolled.Whengraftingwithlowermonomerconcentration,ECscouldgrowonSPU-g-PHEA[poly(2-hydroxyethylacrylate)],SPU-g-PAAm(polyacrylamide),SPU-g-PDMA[poly(2-(dimethylamino)ethylmethacrylate)]orquaternizedSPU-g-PDMAsurfacewithelongatedcellshapes.Hence,thecellcompatibilityofSPUisimprovedandausefulmethodtoconstructacellcompatiblelayeronthepolymersurfacehasbeendeveloped.``
简介:Consideringtheheterogeneityofgeomechanicalmaterials,seismicityduringbrittlerockfailureundercompressiveloadingonthesamplewithanoriginalweakzoneissimulatedbyusingrockfailureprocessanalysiscode(RFPA2D).Therun-throughprocessofweakzone,theformingofnewfaultandassociatedmicro-seismicitiesarestudied.Themodelingdemonstratesthetotalprocessofsourcedevelopmentofearthquakefromdeformation,micro-failuretocollapseandthebehavioroftemporal-spatialdistributionofmicro-seismicities.Thestress,strainandthetemporal-spatialdistributionofmicro-seismicitieslife-likelyportrayedthephenomenaoflocalizationandtemporal-spatialtransitions,whichissimilartothoseobservedinourrealcrust.Also,theresultsobtainedinsimulationsareinagreementwithorsimilartothereportedexperimentalobservations.
简介:
简介:ReportedinthispaperaretheNdisotopiccompositionsofthepre-SinianandSinian-CambriansedimentaryrocksintheXiushuiarea,JiangxiProvince.SignificantdifferencesarenoticedbetweenthemintheirNdisotopicdompositions.Asforthepre-Sinianlightlymetmorphozedsedimentaryrocks,^143Nd/^144Nd=0.512000-0.512214,CNd(T)=-8.04-9.99,andTDM=18332426MaaresuggestedfortheSinian-Cambriansedimentaryrocks.ThesedifferenceswouldreflectthediversityofmaterialsourceforthesedimentaryrocksdepositedbeforeandaftertheSinianperiod.Mantlematerialap-pearstohavebeeninvolvedintheformationofthepre-Siniansedimentaryrockswhilethepost-Siniansedimentaryrocksarecomposedmainlyofrecycleddetritusfromthecontinentalcrust.
简介:ObjectivesTostudythestatusoffibrinolyticinhibitioninpatientsofacutecoronarysyndrome(ACS)complicatedwithtypeⅡdiabetesmellitus(NIDDM)andtoevaluatetheeffectoffibrinolyticinhibitiontotheclinicalprognosis.MethodsTypeⅡdiabetesmellituswasdefinedbyADA1997/WH01998criteria.Thesubjectsweredividedintotreatmentgroupsthatincluded39patientsofACSwith20casesofacutemyocardiacinfarction(AMI),36patientsofACS+NIDOMwith20casesofAMI.Twentycasesofhealthypeoplewererandomizedtocontrolgroup.Theplasmaleveloftissuetypeplasminogenactivator(t-PA),plasminogenactivatorin-hibitortype-1(PAI-1)andplasmaD-dimerweredetectedbyusingelisatechnique.TheindexofstatueinfibrinolysiswasdetectedwiththeplasmalevelofD-dimerandtherateofPAI-1/D-dimerinpercentage.ThisindexwasusedtoevaluatethefibrinolyticinhibitionandtheclinicaloutcomeinallthepatientswithAMIintreatmentgroups.TheclinicaloutcomeinpatientswithAMIconsistedoftherateofreperfusion,theincidencesofreinfarction,severi-ousarrhythmia,pumpfailureanddeathintheearlyperiodofAMI.ResultsTheplasmalevelofPAI-1andD-dimerwashigherinthetwotreatmentgroupsthanthatinthecontrolgroup(P<0.01).TheplasmalevelofPAI-1significantlyhigherinACS+NIDDMpatientsthanthatinACS(P<0.05),buttheplasmalevelofD-dimerraisedfrombasiclevelwassignificanflylowerinACS+NIDDMthanthatinACS(P<0.05).TherateofPAI-1/D-dimerinpercentagewassignificantlyhigherinACS+NIDDMthanthatinACSorincontrolgroup(P<0.01).ForAMIpatientsintwotreatmentgroups,therateofreperfusionafterthethrorabolytictherapywassignifi-candylowerinACS+NIDDMthanthatinACS(P<0.01).TherateofincidencesinpumpfailurewassignificantlyhigherinACS+NIDDMthanthatinACStoo(P<0.05).Themorbidityofseveriousarrhythmia,re-infarctionandthemortalitywerealsohigherinACS+NI
简介:ObjectiveToprovethehypothesisthatthereareassociationsbetweenbodyweightandinsulinresistanceinessentialhypertension,X-syndromesandtype-2diabetes.MethodsWeexaminedthebodymassindex(BMI)andinsulinsensitveindex(ISI)among70hypertensives,39X-syndromes,33type-2diabetesand46controls.ToobservetherelationsbetweenthedegreesofobesityandLSIinallstudygroups.Seventypatientswithhypertension[70hypertensives,48menand22women,meanage(69.3+6.7)years],39X-syndromes[27menand12women,meanage(66.4+6.2)years],33type-2diabetes[21menand12women,meanage(68.7±7.3)years],and46healthycontrols[36menand10women,meanage(66.1+6.6)years].Duringstudyperiod,nopatientsdied.ResultsAllpatientsofhypertensives,X-syndromesandtype-2diabeteshaveinsulinresistance,BMIinallpatientswassingnificantlyhigherthanincontrols.Thelevelsofbloodglucoseandinsulinweresignificantlyhigherinobesehypertensive